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Considering the rate of MVP observed in our cEDS cohort and the knowledge that the mitral valve depends upon collagen for its tensile strength and that myxomatous MVP is characterized by disruption of the collagen layer with expansion of glycosaminoglycans within the middle layer of the valve [123], it is reasonable to consider MVP as a potential clue for cEDS, although the true clinical significance is so far unknown

An a priori power analysis based on results of a prior study 7 and conservative power threshold of 0.95 revealed that a sample size of 2140 was required

Resources and additional reading: The Public Health Law Center, which also provides links to enabling legislation