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In human liver LO2 cells with ethanol-induced oxidative damage, solanesol alleviated hepatotoxicity by activating Keap1/Nrf2/HO-1 and HSF1/Hsp70 signaling pathways, significantly inhibiting the activities of lactate dehydrogenase (LDH) and AST in injured hepatocytes, and blocking ethanol-induced upregulation of MDA and ROS levels and downregulation of glutathione (GSH) levels
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