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We found ferroptotic type I alveolar epithelial cells (CD71 + HOPX + ) and ferroptotic type II alveolar epithelial cells (CD71 + proSP-C + ) are both elevated after ENDS exposure ( Figures 7A, B , Supplementary Figures 1B, C ), suggesting that ENDS induced ferroptosis in epithelial cells, which is partially responsible for alveolar degradation and destruction
Rapid absorption of a large dose through skin contact or oral ingestion can cause severe reactions requiring medical attention and poses risk of fatality in children, animals or highly vulnerable persons
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