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Thirdhand smoke The residue left on furniture, fabrics, and walls reacts with air pollutants to form new carcinogens

In addition, we observe widespread smoking-driven hypermethylation at target regions of the Polycomb repressive complex (OR = 2), which is a well-known aging effect

Chronic use may potentiate cerebrovascular atherosclerosis, elevating long-term stroke risk, particularly in dual users or individuals with underlying risk factors (e.g

In addition, studies have shown that mPTP opening signaling pathways can be inhibited by activating mitophagy [57,58,59], which may represent another mtROS-independent mechanism by which mitophagy inhibits necroptosis