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Assessment of the goiter prevalence in a population is used to define the severity of iodine deficiency, as well as to monitor the long-term impact of sustained salt iodization programs (4, 11)

The investigation into mitochondrial health in these conditions suggests a potential common pathway where viral or bacterial infections lead to mitochondrial damage, resulting in energy production deficits and oxidative stress, which in turn contribute to chronic symptoms

While some findings indicated upregulation of post-meiotic gene expression (33), others emphasized pre-meiotic gene activation [32]

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