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However, present data indicate that IGFBP-1 serves more as a brake on IGF-I action by inhibiting anabolic signaling during nutritional depletion than an accelerator of IGF-I action during overfeeding

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doi: 10.1016/j.soard.2016.08.004

The accumulation of amyloid- (A) is a pathological hallmark of AD, with A being produced through the cleavage of amyloid precursor protein (APP) by -and -secretases, generating peptides of A of varying length (Murphy and LeVine, 2010)