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These data highlighted the benefit of engineering sequence specificity and hydrocarbon stapling to increase the activity of peptide 1907, and also informed the design of our in vitro negative control peptide (2014) with L145E and L152E substitutions, which did not bind FAT in SPR or FP experiments (Table 1 and Fig

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Mechanisms of arsenic Exposure-Induced hypertension and atherosclerosis: an updated overview

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