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Figure 2 Both insoluble fibrils and soluble A oligomers exert potent neurotoxicity by forming plaques and triggering pathological cascades, including synaptic failure, excitotoxicity, changes in membrane permeability, and chronic inflammation ( A hallmark of early AD is systemic impairment of cerebral energy metabolism (304)

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doi:10.2337/dc25-SINT 2

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