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doi: 10.1016/j.chemphyslip.2022.105205 41 DoxakiC.PalikarasK

Sci 118, e2022790118

This role is due to a metabolite of selenocysteine called seleno-cysteinyl (a molecular form of the cysteine moiety) that allows the full gene transcription of Se-GSH-Px (Johansson et al., 2005)

These studies are unable to supply strong evidence to clarify whether their causal link is due to inherent defects, such as selection bias, and other potential confounders (6.6 stars)