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Furthermore, as for cardiac fibroblasts, GLP-1RAs inhibit Ang II and glucose-induced collagen formation via decreasing phospho-NF-B-p65 and phospho-ERK1/2 expression [169] (Figure 2)

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There is some polymorphism for the glutathione synthetase enzyme, so some cases of HAGMA with 5-oxoprolinemia occur later in life under particular metabolic stress, suggesting a previously unrecognized genetic defect [5]