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The two work together because they target different problems

The model also predicted that impairing TCA-dependent NADPH production would elevate the GSSG/GSH ratio and alleviate ER stress, similarly to etomoxir treatment

6 h) activates the Nrf2 signaling pathway via nuclear translocation and upregulates the expression of antioxidant target genes and proteins in primary intestinal epithelial cells (IECs) of chicken embryos [2]