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The intricate relationship between heredity and environmental factors in PsO development and worsening drives home the need to avoid triggers as a means of disease management and identifies possible therapeutic intervention targets [21]
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Studies within mycotoxin research are typically investigating rare outcomes, low exposure frequencies, low effect sizes, etc., which is why they require a bigger sample size
This renders the hypothesis of a Ser111/His108 hTrxR1 dyad for deprotonation of Cys497 hTrxR1 unlikely
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