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Additionally, transcriptomic analysis enables the identification of disease-related biomarkers, which can be used for diagnostic monitoring and treatment, thereby supporting personalized medicine [269]
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(2024) used ELAV1 gene knockout mice to develop an AP model, revealing that ELAV1 upregulates TRAF6 expression, which enhances TLR signaling and promotes M1 macrophage polarization, thereby worsening the course of AP
One cluster analysis of 131 ME/CFS patients diagnosed according to the Fukuda criteria revealed that 34% of patients experienced sympathetic symptoms with dysautonomia (and were associated with more severe disease severity), 5% from sympathetic symptoms alone, 21% parasympathetic, and 40% struggled with sympathovagal balance (331)
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