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However, this mechanism for GLP-1R-induced G s protein activation alone is not sufficient to explain its remarkably sustained cAMP signaling in cells, since the vast majority of GPCRs undergo agonist-dependent desensitization, i.e., G protein decoupling [16]

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Ideally some of the cysteine is eventually converted to sulfate which is used as a conjugate for sulphation, a vital phase 2 detoxification pathway that handles many different toxins including excess oestrogen