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demonstrated that in failing myocardium, stable and detyrosinated microtubules generate viscous forces during diastolic stretch, thereby increasing cardiomyocyte stiffness[6]

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First, they showed that desiccated substrates in either liquid or solid form could be effectively introduced into the solvent-free liquid lipases, indicating that the biofluid phase boundary was accessible to auxiliary agents either by liquidliquid miscibility or solubilization at the liquidsolid interface