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Although breast cancer cell lines exhibit increased resilience to physiologically relevant shear stress compared to normal epithelial cells [223], lamin A/C knockdown exacerbates shear stress-mediated tumor cell death [223], suggesting that elevated lamin A/C levels could aid breast CTCs in withstanding shear forces in the circulation

Guideri, F., Acampa, M., Hayek, Y., and Zappella, M

In artificially induced hyperglycemia, the levels of IL-6, TNF and IL-18 in human plasma increased sharply, which may be related to the mechanism of oxidative stress: Under the condition of hyperglycemia, the uptake of glucose by endothelial cells increases, resulting in excessive production of ROS in mitochondria, which causes oxidative damage and activates the inflammatory signaling cascade in endothelial cells, releasing a large number of inflammatory cytokines into the blood (Esposito et al