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Huxley P, Krayer A, Poole R, Prendergast L, Aryal S, Warner R
Hydrocortisone Component Of Calmurid Hc 390
These indirect implications of RyR2-dependence are supported by findings in skeletal muscle SR vesicles showing LCAC-mediated 45 Ca 2+ release is partially blocked by co-treatment with the ryanodine receptor inhibitor, ruthenium red ( 3 H]ryanodine binding within the SR membranes and markedly increase skeletal muscle ryanodine receptor (RyR1) open probability (Meissner, 1986), affected [ 3 H]ryanodine binding or the rapid 45 Ca 2+ release induced by LCAC treatment (Yamada et al., 2000)
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