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n = 3794) had lower risk in the 2 to 4 years after treatment initiation (post-landmark period) than those who remained in the low-dose range (0.25 to 1.0 mg

We present a detailed picture showing that epithelial-to-mesenchymal transition, cancer metabolic reprogramming, alterations in important signalling pathways, epigenetic aberrations and the tumour microenvironment are crucial drivers of PTC metastasis