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Further, we can assume that the neuromuscular junction function is a needed precondition that BPC 157 therapy (effective in rat injury, given intraperitoneally or in drinking water or topically, at the site of injury) induces the healing of the transected muscle [71], crushed muscle [72], denervated muscle [73], and myotendinous junction [74]

Meanwhile, following a prolonged period of erastin treatment, VDAC2/3 failed to be detected in the cells and such reduced expression resulted in erastin resistance (Yagoda et al., 2007)