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Rather of being generally harmful, inflammation in PD indicates a changing balance between protective clearance systems and maladaptive, self-propagating immune responses (Fig

Target trial emulation: A framework for causal inference from observational data

In the cytoplasm, Keap1 homodimer forms ubiquitin E3 ligase complex with cullin 3 (Cul3), which polyubiquitinates Nrf2 protein leading to its continuous proteasomal degradation ( Figure 3, right)