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Wound-Healing & Repair Pathways GHK-Cu has been shown to accelerate cell migration (keratinocytes and fibroblasts), promote angiogenesis (new blood vessel formation), and improve tissue remodeling after injury

It is proposed to work by binding GHRH receptors on pituitary somatotroph cells, prompting the pituitary to synthesize and release its own growth hormone in a pulsatile pattern, with the body's somatostatin feedback loop still intact so the effect is self-limiting rather than a constant hormone flood

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Disruption in the IFN pathway and defective TLRs lead to SLE