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In more severe patients in whom airway GSH reserves are depleted, this may result in a detrimental cascade with a shift toward GSSG formation and oxidation of thioredoxins, leading to post-translational protein modifications through oxidation of protein-methionine residues and formation of protein disulfides from glutathionylation or other mechanisms (Fig

10.1038/s41419-023-05759-3 221 WangC.YangT.XiaoJ.XuC.AlippeY.SunK.et al (2021)