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First, we assessed levofloxacin persistence in bacterial cultures by pre-exposing the wt and gpoA strains to 20 mM H 2 O 2 for 30 min, followed by treatment with 6 mg/ml levofloxacin for 5 h [8]

Gut barrier damage and endotoxemia: mechanisms and effects Under hyperthermic conditions, superficial blood vessels dilate to dissipate heat, leading to reduced blood flow to visceral organs (especially the intestinal mucosa) in a murine heat stress model, which in turn causes damage and apoptosis of intestinal epithelial cells, increased intestinal epithelial permeability, and impaired intestinal mucosal barrier [17]

doi: 10.1016/s0027-5107(01)00182-8

The blinding of subjects and researchers was reported in four studies (Reference Hashim, Anwar and El-Fatah22,Reference Nasr9,Reference Chandil, Pande and Sen27,Reference Gayatri, Kumar and Kumar28)