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GSH can modulate the activity of various redox-sensitive signaling pathways, including the nuclear factor erythroid 2-related factor 2 (Nrf2) pathway, which regulates the expression of antioxidant and detoxification genes, and the mitogen-activated protein kinase (MAPK) pathway, which regulates cell proliferation, differentiation, and apoptosis

The peptide's efficacy against high-SASP senescent cells proves particularly valuable in the kidney microenvironment, where inflammatory signaling from senescent tubular cells impairs filtration and promotes fibrosis

Extrinsic ageing is caused by external factors like poor lifestyle habits, smoking, alcohol, stress, pollution, and too much sun exposure

Mitochondrial biogenesis facilitates resolution of pyrimidine stress Mitochondrial adaptations promote survival in 5FU-tolerant cells