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In human studies, long-term recreational ketamine use has been associated with structural brain changes, including reduced prefrontal gray matter volume and impaired white matter connectivity, which are potentially linked to chronic low-grade inflammation ( 5.2.2 Mitochondrial dysfunction Ketamine disrupts mitochondrial function by reducing the membrane potential, increasing cytochrome c release, and promoting ROS generation, which collectively induce neuronal apoptosis ( Human studies have identified metabolic abnormalities in ketamine abusers, particularly in the prefrontal cortex, which may reflect mitochondrial dysfunction ( 5.2.3 Dose and frequency dependence The neurotoxic effects of ketamine are highly dependent on its dosage and frequency of use

Potential neuroprotective effects Early studies suggest a role in supporting nerve healing and cognitive health

Cancer history: Growth factor-modulating peptides like Dihexa may warrant avoidance in those with cancer history, though evidence for concern remains theoretical

Mass spectrometry verification of peptide identity to confirm molecular structure and eliminate product authenticity concerns