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In disordered energy homeostasis, circulating nutrient surplus and decreased adipose tissue storage cause the flux of FFAs to the liver, in turn leading to the development of hepatic steatosis [29]

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Current serotonin medications are aimed at increasing central 5-HT neurotransmission in the brain, and there are no approved indications for manipulating peripheral 5-HT, though peripheral 5-HT synthesis is being considered as a potential target for developing pharmacological agents in some disorders ( We are not aware of prior studies reporting low S-5-HT in hypothyroidism or CFS