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Therefore, we identified GSTM2 as an important negative regulator in progression of hepatic steatosis via both its detoxification/antioxidant and inhibition of ASK1-p38/JNK signalling

In conclusion, accelerated translational medicine and advanced non-invasive diagnosis may be gradually bringing fibrosis, a condition associated with a heavy healthcare burden, poor prognosis and systemic disease, into the realm of manageable diseases

Tirzepatide monotherapy evidence is substantial and compelling

IGF2 is thought to be a fetal growth factor, as it is essential for a normal embryonic development and is highly expressed in embryonic and neonatal tissues