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The pathogenesis of OA is driven by accumulative dysregulated pathways, including oxidative stress, autophagy, senescence, secretion of pro-inflammatory cytokines with activation of the NF-B pathway and elevated secretion of the matrix-degrading enzymes metalloproteinases (mainly MMP-3, MMP-13) and aggrecanases (ADAMTS4, ADAMTS5)

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Current Status and Future Directions Some commercial entities have marketed AOD9604 as a research compound or nutraceutical ingredient, though such uses exist in regulatory gray areas