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Dose- and time-dependent increase in GH receptor expression in tendon fibroblasts, potentiating GH response via JAK2 phosphorylation (Kuo et al., 2018)

Although experimental validation of PRODHs role in ferroptosis remains limited across various model systems, a recent study demonstrated that PRODH suppresses tamoxifen-induced ferroptosis in breast cancer cell lines, suggesting a context-dependent antiferroptotic function 102

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They play crucial roles in regulating gene expression critical for cell proliferation, differentiation, and apoptosis[135]