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Importantly, most of the side effects of this drug were gastrointestinal (nausea, diarrhea, etc.), and no serious respiratory side effects were reported (1,11)
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The hypothesis driving its design was straightforward but unverified at the time: that engaging GIP and GLP-1 receptors simultaneously, pathways which are normally co-secreted from the gut following a meal, would produce additive or synergistic effects on insulin secretion, gastric motility, and downstream metabolic markers beyond what either pathway could deliver alone
These antagonists can inhibit the action of IL-1, which, in turn, may restore normal synaptic plasticity and reduce the hyperexcitability associated with seizures (Vezzani et al., 2011)
Among patients without diabetes, the pattern differed from patients with type 2 diabetes: patients on a GLP-1 who maintained a stable weight had a higher osteoporosis risk relative to those who maintained a stable weight on another weight loss medication (22.0% higher for GLP-1 users)
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