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[10] In EDS types I and II, cEDS and clEDS, causative mutations may involved the COL5A1, COL5A2, and tenascin-X genes and are implied to be in the COL1A2 gene

These results are consistent with those of Kochi and Xiao, who suggested that EGCG reduces the severity of liver injury in an experimental model of NAFLD and is associated with lower concentrations of pro-fibrogenic molecules, reactive oxygen species, and pro-inflammatory mediators by modulating TGF/SMAD, PI3K/Akt/FoxO1, and NF-B signaling (28, 56)

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