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These reductions appear to be driven primarily by weight loss, with additional contributions from potential weight-independent mechanisms such as natriuresis, improved endothelial function, and attenuation of vascular inflammation

Because insulin secretion may be challenged in the context of insulin resistance, we next evaluated whether such metabolic dyshomeostasis promoted by -cell selective E2f1 knock-down could be exacerbated upon a metabolic stress such as high fat diet (HFD)

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Stereochemistry: SLU-PP-332 may exhibit more complex stereochemistry, potentially leading to differences in receptor binding and efficacy