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Prolonged exposure of platelets to elevated glucose concentrations can result in alterations to platelet mitochondrial function, including heightened oxygen consumption and an increase in mitochondrial membrane potential, 427 causing excessive production of ROS, and exacerbates platelet activation

Moreover, lifetime AAS dose was associated with a higher coronary atherosclerotic burden [30]

The number needed to treat to prevent a primary endpoint event was 97 over 1 year and 28 over 4 years

"Monotherapy with the once weekly GLP-1 receptor agonist dulaglutide for 12 weeks in Japanese patients with type 2 diabetes: dose-dependent effects on glycaemic control in a randomised, double-blind, placebo-controlled study"