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Alpha-1 antitrypsin inhibits ATP-mediated release of interleukin-1beta via CD36 and nicotinic acetylcholine receptors

The increased PPAR-dependent and decreased NFB-dependent and ER-dependent gene transcription resulting from AKR1C3 inhibition is predicted to inhibit breast cancer cell proliferation, making it a potential target for breast cancer treatment

Inhalation and injection methods may also be effective but should be conducted under medical supervision

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