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References [ 1] Basch, Corey H., et al

This distinction matters enormously for long-term use, where indiscriminate inflammation suppression can compromise immune function and healing capacity

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When insulin engages the -chain, it triggers the insulin receptor substrate (IRS1/2), thereby boosting glucose metabolism (via the PI3K/PKB/AKT pathway), protein synthesis (via the TSC1/2-mTOR pathway), and cell proliferation and differentiation (via the RAS-MAPK-ERK1/2 pathway) (Rebecca et al., 2017