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As expected (70), these diagnoses do not seem to follow predictable Mendelian patterns, suggesting that there are multiple genes and alleles that increase the risk of developing ME/CFS (66, 68)

Subgroup analyses revealed that weight loss was greater in overweight versus obese participants, in unhealthy versus healthy participants, with daily doses 600 mg, and for intervention period shorter than 10 weeks

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In addition, an interaction between USP33 and arrestin3 occurred in an Mdm2-dependent manner (Fig