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Several studies have shown that overexpression of CDC42 from neural stem cell-derived exosomes and Tp53-induced glycolysis and apoptosis regulator (TIGAR) significantly suppresses lipid peroxidation and ferroptosis, thereby promoting neurological recovery

Research reviews also describe BPC-157s protective effects in models involving NSAID-related gastrointestinal lesions, alcohol-related injury, perforated stomach lesions, esophagitis, fistula healing, intestinal inflammation, and other GI injury models

Mild pigmentation concerns 5

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