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However, when C3G was administered to STC-1 cells or NIT-1 cells separately, the NIT-1 cells were unable to release insulin under either low or high glucose conditions

Additionally, the nuclear transcription factor (Foxo1) is phosphorylated upon activation of phosphoinositide- 3-kinaseprotein kinase B/Akt (PI3 K-PKB/Akt), which in turn deactivates molecules to restrict the activity of proapoptotic pathways [66]

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