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On the one hand, dead cells or cellular debris accumulate in the tubular lumen and promote crystal aggregation

These LCACs enhance the ability of RAW264.7 cells to phagocytose lipids by upregulating macrophage receptor with collagenous structure (MARCO), thereby aggravating the accumulation of intracellular lipids, which promotes the formation of foam cells and accelerates the development of atherosclerosis [228]

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It has also shown some potential for mouth-related problems, although the results were mixed [10]