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MBio 11(4)

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In rodent models of acute ischemic stroke induced by middle cerebral artery occlusion (MCAO), semaglutide treatment resulted in a substantial reduction in the volume of the resultant brain infarct by 48%63% and facilitated improved functional recovery ( 4.4 Metabolic dysfunction and other models Semaglutides capacity to ameliorate cognitive deficits induced by a high-fat diet in murine models underscores its potential to directly mitigate the neurological consequences associated with metabolic disorders ( 4.5 Translational limitations and contradictory evidence Despite substantial neuroprotective evidence observed in numerous animal models, it is imperative to recognize significant translational limitations and some contradictory findings

Cell Metab (2012) 16(3):296309