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1) Mechanistic gaps in cellcell crosstalk and non-classical regulatory pathways: Current research focuses on ferroptosis in individual immune cell types (T cells, neutrophils, B cells, and macrophages) but lacks insights into the intercellular crosstalk that amplifies ferroptosis-driven autoimmunity and renal injury in LN

Perhaps in addressing G6PD deficiency, the most promising in the horizon lies in drug development designed to correct the structural defects caused by the various genetic mutations that are becoming increasingly clearer (94)

It is now clear that NAD and/or related metabolites have other roles in the regulation of aging metabolism that are independent of sirtuin activity

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