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Summary Atherosclerotic lesions initiate with endothelial cell dysfunction causing modification of apoB containing lipoproteins (LDL, VLDL, remnants) and infiltration of immune cells, particularly monocytes, into the subendothelial space (Figure 1)
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In addition, ferroptosis promoted by an iron-overloaded environment appears to be detrimental to oocytes or embryos and is also closely related to endometriosis-related infertility
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