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Acute and chronic GI disorders in humans and animal models are characterized by increased ROS production or decreased counteracting antioxidant mechanisms, both of which disrupt redox homeostasis ( Oxidative stress-induced damage in chronic intestinal disorders is associated with mucosal infiltration by activated leukocytes, which produce excessive ROS that overwhelm the tissues antioxidant defenses and perpetuate or exacerbate mucosal inflammation

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For several reasons, however, the expression data remain at best indicative