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However, this function can be compromised during copper overload, as Cu binding to the cysteine residues of GPX4 promotes its ubiquitination and autophagic degradation, thereby disrupting lipid peroxide metabolism and sensitizing cells to ferroptosis [65]

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The broader pathology involves insulin resistance, chronic inflammation, endothelial dysfunction, oxidative stress, dyslipidemia, platelet activation, and arterial stiffness

[DOI] [PMC free article] [PubMed] [Google Scholar] 133.Qin Y., Shi W., Zhuang J., Liu Y., Tang L., Bu J., Sun J., Bei F