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Homocysteine may also react with nitric oxide in the body to form S-nitrosohomocysteine, a compound that causes endothelial damage and reduces the vasodilatory function of blood vessels.2,21 The primary goal in treating hyperhomocysteinemia is to reduce elevated plasma homocysteine levels in order to minimize the risk of neurological and vascular complications

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5 Conclusion This study demonstrates the potential of ceragenins, especially CSA-13, as possible agents for developing new therapies for patients with bloodstream infections

Enzymatic or microbial stability: Biopolymer-based carriers such as resistant starch (RS), inulin-type fructans and pectin-based matrices can resist immediate digestion by human amylases and proteases, which is useful for protecting ingredients until they reach the colon