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A specific list of targets could include direct interference with fungal sulfur uptake via transport inhibitors, blocking EGT biosynthesis via EgtA inhibition (along with Yap1 inhibition), impeding specific steps in ETP biosynthesis (e.g., GliK), inhibition of pantothenic acid, Met and/or Cys biosynthesis or SAH degradation and finally small-molecule interference with enzymes involved in cystathionine formation in fungi

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I., Czobor, P., Citrome, L., Bark, N