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It has been shown that LF-derived peptides mechanisms of hypotensive action involve not only the inhibition of ACE but also interactions with the renin-angiotensin system, the endothelin system, and regulation of gene expression encoding proteins involved in the NO pathway and prostaglandin synthesis (61)

The honest boundary is that even the coherent pairing is a mechanistic hypothesis with no human validation, so the most defensible research posture is to study the single compound at its documented dose first, and treat any combination as a further, clearly-labelled research question rather than a default