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Although several studies supported the role of GIP as an enterogastrone (25, 64, 65), others challenged a role for GIP as an inhibitor of gastric acid secretion under physiological conditions (66, 67)

Its primary researched functions include promoting cell migration (getting healing cells to the injury), down-regulating inflammation, and encouraging the growth of new blood vessels

Essentially, an evolution of BPC 157 without its drawbacks