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The small molecule KY19334, which inhibits the CXXC5/DVL interaction, reactivates Wnt/-catenin signaling, ameliorating liver tissue injury by suppressing pathological phenotypes and enhancing regenerative processes in NASH

Oxidative stress (OxS)/cellular damage Oxidative stress (OxS) is a nonspecific pathophysiological condition that reflects a redox imbalance between increased production of ROS (free radicals) and the inability of antioxidant defenses to neutralize the reactive intermediates or to repair the ensuing damage 179,180

doi: 10.1016/S1569-1993(03)00097-3

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