Summary and outlook In recent years, substantial clinical and basic research has advanced our understanding of heat stress-induced liver injury, yet its complex pathogenesischaracterized by interconnected mechanisms including hyperthermia-induced direct hepatocyte damage, oxidative stress, inflammatory cascades, and diverse programmed cell death modes (apoptosis, necroptosis, pyroptosis, ferroptosis)remains incompletely elucidated
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Our results showed that the HT treatment increased MDA content and O 2 production rate in cucumber leaves, which was significantly decreased in the HT+GSH plants (Fig
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